Short answer
Eliminate irritating foods, perform FGDS and tests for Helicobacter pylori. For acute symptoms, consult a gastroenterologist.
Inflammation of the gastric mucosa with indigestion
The mechanism in three steps, and where you can intervene.
Trigger
Helicobacter pylori, NSAIDs, alcohol, stress, spicy food
Mechanism
Damage to the mucosal barrier → inflammation → impaired secretion of hydrochloric acid and pepsin
Symptom
Epigastric pain, heartburn, nausea, feeling of fullness after eating
Answer 3 questions to see likely causes and a baseline lab list.
How long has this been going on?
Is there marked fatigue?
Is stress high?
Likely causes
What to check
We will show a preliminary result before registration
Create an action plan in one click with a checklist, reminders, and a timeline.
Eliminate irritating foods, perform FGDS and tests for Helicobacter pylori. For acute symptoms, consult a gastroenterologist.
A baseline panel for the most common causes.
Identification of bacteria - the main cause of gastritis
Visual assessment of the mucosa and histology
Detection of anemia in chronic gastritis
Assessment of iron reserves in atrophic gastritis
How each marker relates to gastritis.
The main cause is Helicobacter pylori infection (80% of cases). Long-term use of NSAIDs (aspirin, ibuprofen), alcohol abuse, autoimmune processes and bile reflux also damage the mucosa. Stress and irregular nutrition aggravate the condition.
The H. pylori breath test is the gold standard for diagnosis. FGDS allows you to assess the extent of the lesion and take a biopsy. A general blood test reveals anemia, and ferritin is a hidden iron deficiency in the atrophic form. Additionally, pepsinogens I/II may be required.
For persistent heartburn, pain on an empty stomach or after eating, black stools (melena) or vomiting “coffee grounds”. Urgent consultation is needed if there is sudden weight loss, difficulty swallowing, or anemia of unknown origin.
Self-medication with soda for heartburn aggravates damage to the mucous membrane. Long-term use of PPIs without H. pylori diagnosis masks symptoms. Ignoring the diet provokes relapses. Refusal of FGDS due to fear of the procedure leads to late diagnosis of atrophy.
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Основано на структурированном медицинском подходе, не на хаотичных советах из интернета.
Не заменяет очную консультацию и постановку диагноза врачом.
Срочные симптомы требуют немедленного обращения за медицинской помощью.
Acute gastritis, when the cause is eliminated (H. pylori, NSAIDs), is completely curable. Chronic forms require lifelong diet and control. Atrophic gastritis is irreversible, but can be corrected.
Table No. 1 according to Pevzner: pureed soups, steamed dishes, exclusion of spicy/fried/coffee. In the hypoacid form - secretion-stimulating food (meat broths).
Leads to ulcers, mucosal atrophy, B12-deficiency anemia. Long-term inflammation increases the risk of stomach cancer by 5-6 times.
Exactly - only at FGDS. The ulcer gives “hungry” pain at night and can be complicated by bleeding. Gastritis often manifests itself as discomfort after eating.
Yes - atrophic gastritis is often asymptomatic. After 40 years, FGDS is recommended once every 2-3 years, especially if there is a family history of stomach cancer.
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